ScienceDaily (Mar. 29, 2012) — Hunger has always threatened humankind. This makes it unsurprising that human bodies attempt to store all surplus nourishment in adipose tissue. In developed countries, this life-saving craving is turning into a problem and obesity -- adiposity -- is turning into a danger. Why, though, does excess fat the body ill? How does fat trigger diabetes? And can these superfluous fat reserves be turned into warmth and just as well burnt?
Marcel Scheideler from the Technical University of Graz has examined these questions intensively in a particular project of the Austrian genomic research programme GEN-AU; and he discovered that microRNAs are a part of the solution. It has been known for a while that these molecules, which are produced by the human body, play an important role in gene regulation and thus contribute to cancer genesis. Indeed, this is a research area to which Scheideler has already made valuable contributions as part of an interdisciplinary research team (1). The fact that microRNAs could also contribute to obesity and diabetes, however, is a new insight, to which Scheideler and his team have contributed significantly recently (2-4). The topic of RNA and adiposity thus played an important role at the current conference "9 Years of the GEN-AU Programme" in Innsbruck. The participating research teams were managed by the local Innsbruck company CEMIT.
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